Guillain-Barre Syndrome
- Guillian-Barre Syndrome (GBS), or acute inflammatory demyelinating polyradiculoneuropathy, affects nerve roots and peripheral nerves leading to motor neuropathy and flaccid paralysis.
- Common diagnostic features of GBS include motor weakness, mild sensory symptoms,& autonomic dysfunction.
- Recovery usually begins 2 to 4 weeks after plateau of disease process.
- Clinical presentation: characterized by a rapidly evolving, relatively symmetrical ascending weakness or flaccid paralysis. Motor impairment may vary from mild weakness of distal lower-extremity musculature to total paralysis of the peripheral, axial, facial, and extraocular musculature.
- Prognosis: Approximately 80% become ambulatory within 6 months of onset of symptoms. The most common long-term deficits are weakness of the anterior tibial musculature, and less often weakness of the foot and hand intrinsics, quadriceps, and gluteal musculature.
Source: Umphred, D.A. (2001). Neurological Rehabilitation. 4th Ed. U.S.A.: Mosby, Inc.
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- Guillian-Barre Syndrome (GBS), or acute inflammatory demyelinating polyradiculoneuropathy, affects nerve roots and peripheral nerves leading to motor neuropathy and flaccid paralysis.
- Common diagnostic features of GBS include motor weakness, mild sensory symptoms,& autonomic dysfunction.
- Recovery usually begins 2 to 4 weeks after plateau of disease process.
- Clinical presentation: characterized by a rapidly evolving, relatively symmetrical ascending weakness or flaccid paralysis. Motor impairment may vary from mild weakness of distal lower-extremity musculature to total paralysis of the peripheral, axial, facial, and extraocular musculature.
- Prognosis: Approximately 80% become ambulatory within 6 months of onset of symptoms. The most common long-term deficits are weakness of the anterior tibial musculature, and less often weakness of the foot and hand intrinsics, quadriceps, and gluteal musculature.
Source: Umphred, D.A. (2001). Neurological Rehabilitation. 4th Ed. U.S.A.: Mosby, Inc.